ACTA NEUROPHARMACOLOGICA ›› 2020, Vol. 10 ›› Issue (3): 21-26.DOI: 10.3969/j.issn.2095-1396.2020.03.004
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GAO Yun-xia，ZHANG Yuan-yuan
Online:2020-06-26
Published:2021-05-14
Contact:
张元元，女，讲师，博士；研究方向：眼科疾病及抗肿瘤药物筛选；E-mail：yuanyuanzhangyyz@163.com
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GAO Yun-xia, ZHANG Yuan-yuan. The Role of Endoplasmic Reticulum Stress in Senile Macular Degeneration[J]. ACTA NEUROPHARMACOLOGICA, 2020, 10(3): 21-26.
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URL: http://actanp.hebeinu.edu.cn/EN/10.3969/j.issn.2095-1396.2020.03.004
| [1] 杨焕娟, 关瑞娟, 李凌. 年龄相关性黄斑病变流行病学研究进展[J]. 高原医学杂志, 2019, 29(3): 60-63. [2] 闫明. 年龄相关性黄斑变性的相关机制与临床研究[D]. 武汉: 南方医科大学, 2018. [3] 庄娟. 内质网应激[J]. 生物学教学, 2012, 37(12): 2-3. [4] 宋乐乐, 刘浩, 蒋琛琛. 内质网应激与自噬信号通路研究进展[J]. 蚌埠医学院学报, 2014, (5): 687-690. [5] Richard Jager, Mathleu JM Bertrand, Adrlenne Gorman, et al. The unfolded protein response at the crossroads of cellular life and death during endoplasmic reticulum stress[J]. Biol Cell, 2012, 104 (5): 259-270. [6] Adrienne M Gorman, Sandra J M Healy, Richard Jager, et al. Stress management at the ER: regulators of ER stress-induced apoptosis[J]. Pharmacol Ther, 2012, 134(3): 306-316. [7] 徐静尊, 鲁敏. 内质网应激与自噬及其交互作用影响内皮细胞凋亡[J]. 中国生物化学与分子生物学报, 2019, 35(3): 240-250. [8] Ryan A V Bell, Lynn A Megeney. Evolution of caspase-mediated cell death and differentiation: twins separated at birth[J]. Cell Death Differ, 2017, 24(8): 1359-1368. [9] 陈娜子, 姜潮, 李校堃. 内质网应激与疾病[J].中国生物工程杂志, 2016, 36(1): 76-85. [10] Ana Maria Snchez, Javier Martínez-Botas, Sophie Malagarie-Cazenave, et al. Induction of the endoplasmic reticulum stress protein GADD153/CHOP by capsaicin in prostate PC-3 cells: a microarray study[J]. Biochem Biophys Res Commun, 2008, 372(4): 785-791. [11] Helene Zinszner, Masahiko Kuroda, Wang Xiao-zhong, et al. CHOP is implicated in programmed cell death in response to impaired function of the endoplasmic reticulum[J]. Genes Dev, 1998, 12(7): 982-995. [12] Fu Hai-ying, Ken-ichiro Okada, Liao Yu-lin, et al. Ablation of C/EBP homologous protein attenuates endoplasmic reticulum-mediated apoptosis and cardiac dysftinction induced by pressure overload[J]. Circulation, 2010, 122(4): 361-369. [13] Decio L Eizirik, Alessandra K Cardozo, Miriam Cnop. The role for endoplasmic reticulum stress in diabetes mellitus[J]. Endocr Rev, 2008, 29(1): 42-61. [14] 邹文军, 文峰. 抗氧化营养素与老年性黄斑变性的防治[J]. 眼科学报, 2007, 23(4): 193-198. [15] Stephen Beatty, Hui-Hiang Koh, David Henson, et al. The role of oxidative stress in the pathogenesis of age—related macular degeneration[J]. Surv Ophthalmol, 2000, 45(2): 115-134. [16] Marco Zarbin. Current concepts in the pathogenesis of age—related macular degeneration[J]. Arch OphthMmol, 2004, 122(4): 598-614. [17] Li Gang, Christopher Scull, Lale Ozcan, et al. NADPH oxidase links endoplasmic reticulum stress, oxidative stress, and PKR activation to induce apoptosis[J]. J Cell Biol, 2010, 191(6): 1113-1125. [18] 邱艳丽, 李巧稚, 郑亚琴, 等. 内质网应激与氧化应激:恶性循环还是双刃剑?[J]. 实用药物与临床, 2016, 19(8): 1037-1041. [19] Malhotra J D, Kaufman R J. Endoplasmic reticulum stress and oxidative stress: a vicious cycle or a double-edged sword[J]. Antioxid Redox Signal, 2007, 9(12): 2277-2293. [20] Stewart Siyan Cao, Randal J Kaufman. Endoplasmic reticulum stress and oxidative stress in cell fate decision and human disease[J]. Antioxid Redox Signal, 2014, 21(3): 396-413. [21] Yu W, Liu X, Feng L, et al. Glycation of paraoxonase 1 by high glucose instigates endoplasmic reticulum stress to induce endothelial dysfunction in vivo[J]. Sci Rep, 2017, 7: 45827. [22] Atsuko Nakatsuka, Jun Wada, Izuml Iseda, et al. Visceral adipose tissue-derived serine proteinase inhibitor inhibits apoptosis of endothelial cells as a ligand for the cell-surface GRP78/voltage-dependent anion channel complex[J]. Circ Res, 2013, 112(5): 771-780. [23] Josefa Girona, Cella Rodríguez-Borjabad, Dalana Ibarretxe, et al. The circulating GRP78/Bi P is a marker of metabolic diseases and atherosclerosis: Bringing endoplasmic reticulum stress into the clinical scenario[J]. JClin Med, 2019, 8(11): E1793. [24] 罗明, 余卫强. 脂肪细胞内质网应激反应蛋白与肥胖的关系[J]. 中国临床解剖学杂志, 2020, 38(2): 235-238. [25] Justyna Janikiewicz, Katarzyna Hanzelka, Kamil Kozinski, et al. Islet beta-cell failure in type 2diabetes-Within the network of toxic lipids[J]. Biochem Biophys Res Commun, 2015, 460 (3): 491-496. [26] Louise Deldicque, Patrice D Cani, Andrew Philp, et al. The unfolded protein response is activated in skeletal muscle by high-fat feeding:Potential role in the downregulation of protein synthesis[J]. Am J Physiol Endocrinol Metab, 2010, 299(5): E695-705. [27] Louise Deldicque, Patrice D Cani, Nathalie M Delzenne, et al. Endurance training in mice increases the unfolded protein response induced by a high-fat diet[J]. J Physiol Biochem, 2013, 69(2): 215-225. [28] Louise Deldicque, Karen Van Proeyen, Marc Francaux, et al. The unfolded protein response in human skeletal muscle is not involved in the onset of glucose tolerance impairment induced by a fat-rich diet[J]. Eur J Appl Physiol, 2011, 111(1): 1553-1558. [29] 张羽航, 鲍时翔, 郑学勤, 等. 脂肪酸脱饱和酶的研究进展[J]. 生物技术通报, 1998, (4): 109-109. [30] 张喆. 运动改善内质网应激与肥胖相关疾病的研究进展[J]. 中国体育科技, 2016, 52(2): 80-84. [31] Adina D Mihai, Martin Schröder. Glucose starvation and hypoxia, but not the saturated fatty acid palmitic acid or cholesterol,activate the unfolded protein responsein 3T3-F442A and 3T3-L1adipocytes[J]. Adipocyte, 2015, 4(3): 188-202. [32] He Y, Sun S, Sha H, et al. Emerging roles for XBP1, as UPe Rtranscription factor[J]. Gene Expr, 2010, 15(1): 13-25. [33] 刘宝琴, 王华芹. 内质网应激与未折叠蛋白反应的研究进展[J]. 中华肿瘤防治杂志, 2010, 17(11): 869-872. [34] 张海燕, 刘宝琴, 邓娓娓, 等. GRP78在蛋白酶体抑制剂诱导甲状腺癌细胞凋亡中的作用[J]. 中华肿瘤防治杂志, 2009, 16(18): 1379-1382. [35] 郭腾飞, 轩青霞, 吴玉丹, 等. GRP78—ATF6—CHOP通路相关分子在葡聚糖硫酸钠诱导的小鼠结肠炎中的表达[J]. 郑州大学学报, 2019, 6: 806-811. [36] 丰梅,付凌玲,张伟华, 等. 内质网应激调控细胞自噬与凋亡[J]. 中国细胞生物学学报, 2018, 40(3): 455-462. [37] 扎西英派, 张涛杰, 霍生东, 等. 葡萄糖调节蛋白GRP78研究进展[J]. 西北民族大学学报, 2015, 36(1): 48-58. [38] Peter Walter, David Ron. The unfolded protein response: from stress pathway to homeostatic regulation[J]. Science, 2011, 334(6059): 1081-1086. [39] Heather P Harding, Marcella Calfon, Fumlhlko Urano, et al. Transcriptional andtranslational control in the Mammalian unfolded protein response[J]. Annu Rev Cell Dev Biol, 2002, 18(1): 575-599. [40] Min Ni, Hui Zui, Shiuan Shannon Wey, et al. Regulation of PERKsignaling andleukemia cell survival by a novel cytosolic isoform of the UPRregulator GRP78/Bip[J]. PLos One, 2009, 4(8): 6868-6882. [41] Mina Thon, Toru Hosoi, Koichiro Ozawa. Dehydroascorbic acid-induced endoplasmic reticulum stressand leptin resistance in neuronal cells[J]. Biochem Biophys Res Commun, 2016, 478(2): 716-720. |
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